Computerized analysis of the CTG
analysis of the fetal heart rate provides real time alerts
to aid intrapartum management and has shown to improve the accuracy of clinicians when analyzing CTGs.
leads the technology in CTG analysis
, with extensive publishing on reference journals and the involvement of top authors from the field.
estimation of uterine contractions, of fetal heart rate baseline, identification of accelerations, decelerations, and quantification of short- and long-term variability.
Visual and Sound Alerts
provide just visual messages.
In this example they may help managing oxytocin use and suggest searching for reactivity signs.
have also a sound alert and some are emergency cases like the one shown below.
At any time a user may open a window with
more detail on the analysi
Both quantitative and Graphical results are available
The graphical results contain the estimated baseline and the calculations
for accelerations, decelerations and uterine contractions.
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Related to conditions that are not usually associated with fetal hypoxia but may deserve some action on the part of health professionals.
The “Excessive uterine contractions” alert should motivate the evaluation of an eventual oxytocin induction/augmentation of labour and suggests careful monitoring for the appearance of hypoxemia-related changes in the fetal heart rate.
Yellow and Orange Alerts
These alerts signify the occurrence of fetal heart rate events that are not normal, but which by themselves are also not specific of fetal hypoxia.
can be caused by drugs administered to the mother or by fetal hypoxemia.
Possible causes for isolated
are maternal hyperthermia, intra-uterine infection, drugs administered to the mother and hypoxemia.
On their own, they do not suggest fetal hypoxia, but should motivate the attempt to correct/treat the underlying situation.
is very rare and can be caused by maternal hypothermia or fetal heart dysrhythmias.
can be caused by fetal head compression, transient fetal hypertension caused by umbilical cord compression or transient hypoxemia.
The simultaneous appearance of
suggests some hypoxic risk, and a lower threshold for intervention in probably appropriate in these situations.
As hypoxemia is a possible cause of these changes efforts should be made to uncover and to correct causal or contributing situations (mother lying on her back, maternal hypotension, uterine hyper contractility).
If CTG changes do not revert rapidly, fetal ECG with ST analysis or FBS can be considered.
These alerts are associated with a high probability of central fetal hypoxia, and should elicit an action from health professionals for the immediate reversal of a cause of hypoxia or the rapid removal from the intra-uterine environment.
Other factors must also be taken into account, such as gestational age, maternalhyperthermia, administered drugs, intra-uterine growth restriction, previous CTG patterns, etc.